ProQuest Subject Index - Physiology at TEMPLE UNIVERSITY

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You are viewing titles for TEMPLE UNIVERSITY in the Physiology available through the UMI Dissertations & Thesis Gradwoorks site

The role of arginase in nitric oxide mediated contractile function and survival in normal and diseased myocardium	The protective phenotype associated with exercise training on the hypertensive myocardium

Hematopoietic lineage cell-specific protein-1 (HS1) is a functionally important signaling molecule in platelet activation	Modulation of the cannabinoid CB1 and CB2 receptor activation in central nervous system disorders

Ethnic variations in body image and physical activity in women's cardiovascular health	Comparison of a low carbohydrate and calorie-restricted diet on weight loss, morphometric variables, and diet acceptability

Altered regulation of contractile reserve in failing myocardium	The effect of exercise on the cardiac phenotype of the female Spontaneously Hypertensive Rat

Cardiac stem cells generate new cardiac myocytes during normal cardiac development and pathological hypertrophy	Perfluorochemical augmented intratracheal delivery of antioxidant enzymes and genes to attenuate oxidative stress-induced lung and respiratory muscle alterations

Role of protease-activated receptors in platelet activation	Molecular mechanisms underlying differential regulation of platelet dense granule secretion by protein kinase C delta

The effects of high repetition low force motion on tendon integrity and motor behavior in an animal model of work-related musculoskeletal disorders	Molecular physiology of thromboxane A2 generation in platelets

Molecular physiology of novel class of protein kinase c isoforms in platelets	Re-expression of T-type calcium channels minimally affects cardiac contractility and activates pro-survival signaling pathways in the myocardium

Regulation of protein kinases (Syk and PKC Zeta) in platelets	Increasing myocyte contractility exacerbates cardiac injury and pump dysfunction and ablation of phosphorylation site in RyR2 at Ser-2808 has no protection against cardiac dysfunction progression after myocardial infarction

Repair of the injured adult heart involves resident cardiac stem cell derived new myocytes	In vivo and in vitro interactions of oxidative stress and laminar shear stress on vascular endothelial growth factor-mediated endothelial nitric oxide synthase activity

Does calcium influx through T-type calcium channel induce cardiomyocyte proliferation?